Publications

(abstract)
 
(2015)
 
Bertero, A., Madrigal, P., Galli, A., Hubner, NC., Moreno, I., Burks, D., Brown, S., Pedersen, RA., Gaffney, D., Mendjan, S., Pauklin, S., Vallier, L. (2015). Activin/nodal signaling and NANOG orchestrate human embryonic stem cell fate decisions by controlling the H3K4me3 chromatin mark. Genes Dev. 29(7):702-17 (abstract)
 
Faial, T., Bernardo, AS., Mendjan, S., Diamanti, E., Ortmann, D., Gentsch, GE., Mascetti, VL., Trotter, MW., Smith, JC., Pedersen, RA. (2015). Brachyury and SMAD signalling collaboratively orchestrate distinct mesoderm and endoderm gene regulatory networks in differentiating human embryonic stem cells. Development. 142(12):2121-35 (abstract)
 
(2014)
 
Mendjan, S., Mascetti, VL., Ortmann, D., Ortiz, M., Karjosukarso, DW., Ng, Y., Moreau, T., Pedersen, RA. (2014). NANOG and CDX2 pattern distinct subtypes of human mesoderm during exit from pluripotency. Cell Stem Cell. 15(3):310-25 (abstract)
 
(2012)
 
Pedersen, RA., Mascetti, V., Mendjan, S. (2012). Synthetic organs for regenerative medicine. Cell Stem Cell. 10(6):646-7 (abstract)
 
Sun, B., Ito, M., Mendjan, S., Ito, Y., Brons, IG., Murrell, A., Vallier, L., Ferguson-Smith, AC., Pedersen, RA. (2012). Status of genomic imprinting in epigenetically distinct pluripotent stem cells. Stem Cells. 30(2):161-8 (abstract)
 
(2009)
 
Vallier, L., Mendjan, S., Brown, S., Chng, Z., Teo, A., Smithers, LE., Trotter, MW., Cho, CH., Martinez, A., Rugg-Gunn, P., Brons, G., Pedersen, RA. (2009). Activin/Nodal signalling maintains pluripotency by controlling Nanog expression. Development. 136(8):1339-49 (abstract)
 
(2007)
 
Mendjan, S., Akhtar, A. (2007). The right dose for every sex. Chromosoma. 116(2):95-106 (abstract)
 
(2006)
 
Mendjan, S., Taipale, M., Kind, J., Holz, H., Gebhardt, P., Schelder, M., Vermeulen, M., Buscaino, A., Duncan, K., Mueller, J., Wilm, M., Stunnenberg, HG., Saumweber, H., Akhtar, A. (2006). Nuclear pore components are involved in the transcriptional regulation of dosage compensation in Drosophila. Mol Cell. 21(6):811-23 (abstract)
 

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